Association between Asprosin and Clusterin in patients with Myocardial infarction.

Document Type : Original Article

Authors

1 , Chemistry Department, College of Science, Mustansiriyah University , Baghdad – Iraq.

2 Missions and cultural Relations department, AI-Iraqia University, Baghdad- Iraq.

3 Biology Department, College of Science, Mustansiriyah University , Baghdad – Iraq.

4 College of medicine, Alnahrain university, Baghdad – Iraq.

Abstract

Background: The most common cause of acute myocardial infarction (AMI) is still plaque rupture. Elevation of ST segments. Myocardial infarction (STEMI) and elevation without ST-segment. There are two forms of AMI, Myocardial Infarction (NSTEMI) and AMI, each with unique clinical features. Asprosin is a newly discovered fasting-induced glucogenic adipokine that stimulates the liver to release glucose into the bloodstream. It has been linked to metabolic disorders like polycystic ovarian syndrome and type II diabetes mellitus. Most biological fluids contain the heterodimeric glycoprotein clusterin, which is produced by a variety of tissues. Based on clusterin's distribution and in vitro properties, several physiological roles have been suggested. A notable and distinctive characteristic of clusterin is its induction in various disease states, including but not limited to glomerulonephritis, polycystic kidney disease, renal tubular injury, and neurodegenerative conditions such as Alzheimer's disease, atherosclerosis, and myocardial infarction. 

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